1. Regulation of Ovulation Rate --;2. Dynamics of Follicle Development in the Human Ovary --;3. The Molecular Basis of Ovarian Cell Death --;4. Polycystic Ovary Syndrome (PCOS): The Possible Roles of Apoptosis in Human Granulosa Cells --;5. Potential Role of the Renin-Angiotensin System in Polycystic Ovaries --;6. Dysregulation of Androgen Secretion and Steroid Metabolism in Polycystic Ovary Syndrome --;7. Adrenocortical Dysfunction in the Polycystic Ovary Syndrome --;8. Insulin Resistance in the Polycystic Ovary Syndrome --;9. Beta-Cell Function in Polycystic Ovary Syndrome --;10. Luteinizing Hormone and Growth Factor Control of the Thecal Cell --;11. Comparative Androgen Production from Theca Cells of Normal Women and Women with Polycystic Ovaries --;12. Development of a Human Thecal Tumor Cell Model: Regulation of Steroidogenesis and Enzyme Expression --;13. Development of Human Granulosa Cell Lines --;14. Suppression of Aromatase Activity in Polycystic Ovary Syndrome --;15. The Insulin-Like Growth Factor (IGF) System in Human Ovary and Its Relevance to Polycystic Ovarian Syndrome --;16. Hyperinsulinemic Androgenism: A Pathophysiologic Paradox --;17. Hypothalamic-Pituitary Dynamics in Polycystic Ovary Syndrome --;18. Aspects of Ovulation Induction in Polycystic Ovary Syndrome --;19. Polycystic Ovary Syndrome, Fertilization, and Early Embryonic Development --;20. In Vitro Fertilization in Polycystic Ovary Syndrome --;21. Surgical Approaches to Ovulation Induction: The Laparoscopic Experience --;22. Androgens, Lipids, Insulin Resistance, and Cardiovascular Risk --;23. A Unifying Concept for Polycystic Ovary Syndrome --;Author Index.
SUMMARY OR ABSTRACT
Text of Note
The term polycystic ovary syndrome (peOS) is meant to describe a clinical endocrinopathy characterized by menstrual irregularity and evidence of hyperandrogenism.